Arteriosclerosis is not an inevitable eonsequenee of age, as formerly supposed, but a reaetiv disease. It ean be prevented by elimination of the pathogenic faetors and regression of the lesions of the wall strueture by treatment is possible. Our experiments with animal models demonstrate that the aeeeierated metabolism of the arterial wall eells, indueed by very many kinds of pathogenic faetors, of risk faetors, is the real pathologie proeess,...
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